PRL2 inhibition elevates PTEN protein and ameliorates progression of acute myeloid leukemia
Author:
Funder
National Institutes of Health
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Link
https://insight.jci.org/articles/view/170065/files/pdf
Reference51 articles.
1. Regulatory Mechanisms and Novel Therapeutic Targeting Strategies for Protein Tyrosine Phosphatases
2. Molecular Pathways: Targeting Protein Tyrosine Phosphatases in Cancer
3. PRL phosphatases as potential molecular targets in cancer
4. Phosphatase of regenerating liver: a novel target for cancer therapy
5. Physiological and oncogenic roles of thePRLphosphatases
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1. Targeting PRL phosphatases in hematological malignancies;Expert Opinion on Therapeutic Targets;2024-04-02
2. Protein Tyrosine Phosphatase PRL-3: A Key Player in Cancer Signaling;Biomolecules;2024-03-12
3. Inhibition of PRL2 Upregulates PTEN and Attenuates Tumor Growth in Tp53-deficient Sarcoma and Lymphoma Mouse Models;Cancer Research Communications;2024-01-02
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