Requirement for Ca2+/calmodulin–dependent kinase II in the transition from pressure overload–induced cardiac hypertrophy to heart failure in mice-Reference-Cited by-同舟云学术

Requirement for Ca2+/calmodulin–dependent kinase II in the transition from pressure overload–induced cardiac hypertrophy to heart failure in mice

Published:2009-05-01 Issue:5 Volume:119 Page:1230-1240
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Ling Haiyun,Zhang Tong,Pereira Laetitia,Means Christopher Kable,Cheng Hongqiang,Gu Yusu,Dalton Nancy D.,Peterson Kirk L.,Chen Ju,Bers Donald,Heller Brown Joan

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference60 articles.

1. Fractional SR Ca release is regulated by trigger Ca and SR Ca content in cardiac myocytes

2. Phosphorylation Modulates the Function of the Calcium Release Channel of Sarcoplasmic Reticulum from Cardiac Muscle

3. Site-specific phosphorylation of a phospholamban peptide by cyclic nucleotide- and Ca2+/calmodulin-dependent protein kinases of cardiac sarcoplasmic reticulum

4. Unique phosphorylation site on the cardiac ryanodine receptor regulates calcium channel activity

5. Ca2+/Calmodulin-Dependent Protein Kinase II Phosphorylation Regulates the Cardiac Ryanodine Receptor

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