Prolyl hydroxylase 3 (PHD3) is essential for hypoxic regulation of neutrophilic inflammation in humans and mice-Reference-Cited by-同舟云学术

Prolyl hydroxylase 3 (PHD3) is essential for hypoxic regulation of neutrophilic inflammation in humans and mice

Published:2011-03-01 Issue:3 Volume:121 Page:1053-1063
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Walmsley Sarah R.,Chilvers Edwin R.,Thompson Alfred A.,Vaughan Kathryn,Marriott Helen M.,Parker Lisa C.,Shaw Gary,Parmar Selina,Schneider Martin,Sabroe Ian,Dockrell David H.,Milo Marta,Taylor Cormac T.,Johnson Randall S.,Pugh Christopher W.,Ratcliffe Peter J.,Maxwell Patrick H.,Carmeliet Peter,Whyte Moira K.B.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference42 articles.

1. Cyclin-dependent kinase inhibitors enhance the resolution of inflammation by promoting inflammatory cell apoptosis

2. Hypoxia-induced neutrophil survival is mediated by HIF-1α–dependent NF-κB activity

3. Cellular adaptation to hypoxia: O2‐sensing protein hydroxylases, hypoxia‐inducible transcription factors, and O2‐regulated gene expression

4. HIF-1α Is Essential for Myeloid Cell-Mediated Inflammation

5. HIF-1α expression regulates the bactericidal capacity of phagocytes

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