BCL11A deletions result in fetal hemoglobin persistence and neurodevelopmental alterations-Reference-Cited by-同舟云学术

BCL11A deletions result in fetal hemoglobin persistence and neurodevelopmental alterations

Published:2015-05-04 Issue:6 Volume:125 Page:2363-2368
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Basak Anindita,Hancarova Miroslava,Ulirsch Jacob C.,Balci Tugce B.,Trkova Marie,Pelisek Michal,Vlckova Marketa,Muzikova Katerina,Cermak Jaroslav,Trka Jan,Dyment David A.,Orkin Stuart H.,Daly Mark J.,Sedlacek Zdenek,Sankaran Vijay G.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference24 articles.

1. The Switch from Fetal to Adult Hemoglobin

2. Anemia: progress in molecular mechanisms and therapies

3. Human Fetal Hemoglobin Expression Is Regulated by the Developmental Stage-Specific Repressor BCL11A

4. Developmental and species-divergent globin switching are driven by BCL11A

5. Bcl11a is essential for lymphoid development and negatively regulates p53

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