Integrated compensatory network is activated in the absence of NCC phosphorylation-Reference-Cited by-同舟云学术

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Integrated compensatory network is activated in the absence of NCC phosphorylation

Published:2015-04-20 Issue:5 Volume:125 Page:2136-2150
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Grimm P. Richard,Lazo-Fernandez Yoskaly,Delpire Eric,Wall Susan M.,Dorsey Susan G.,Weinman Edward J.,Coleman Richard,Wade James B.,Welling Paul A.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference101 articles.

1. The thiazide-sensitive Na+-Cl−cotransporter: molecular biology, functional properties, and regulation by WNKs

2. Primary structure and functional expression of a cDNA encoding the thiazide-sensitive, electroneutral sodium-chloride cotransporter.

3. Phenotype Resembling Gitelman's Syndrome in Mice Lacking the Apical Na+-Cl− Cotransporter of the Distal Convoluted Tubule

4. Clarification of the site of action of chlorothiazide in the rat nephron.

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