Inhibition of mitochondrial fragmentation diminishes Huntington’s disease–associated neurodegeneration
Author:
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Reference91 articles.
1. Huntington's disease: from molecular pathogenesis to clinical treatment
2. A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes
3. Mitochondrial fission and cristae disruption increase the response of cell models of Huntington's disease to apoptotic stimuli
4. Mutant huntingtin binds the mitochondrial fission GTPase dynamin-related protein-1 and increases its enzymatic activity
5. Oxidative Stress in Huntington's Disease
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