Epithelial HIF-1α/claudin-1 axis regulates barrier dysfunction in eosinophilic esophagitis
Author:
Funder
NIH NIDDK
American Partnership for Eosinophilic Disorders APFED
NIH NCATS
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Link
https://www.jci.org/articles/view/126744/files/pdf
Reference53 articles.
1. Eosinophilic Esophagitis Is a Late Manifestation of the Allergic March
2. Coordinate Interaction between IL-13 and Epithelial Differentiation Cluster Genes in Eosinophilic Esophagitis
3. Genome-wide association analysis of eosinophilic esophagitis provides insight into the tissue specificity of this allergic disease
4. Epithelial origin of eosinophilic esophagitis
5. Solute carrier family 9, subfamily A, member 3 (SLC9A3)/sodium-hydrogen exchanger member 3 (NHE3) dysregulation and dilated intercellular spaces in patients with eosinophilic esophagitis
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