Intracerebral transplantation of mesenchymal stem cells into acid sphingomyelinase–deficient mice delays the onset of neurological abnormalities and extends their life span-Reference-Cited by-同舟云学术

Intracerebral transplantation of mesenchymal stem cells into acid sphingomyelinase–deficient mice delays the onset of neurological abnormalities and extends their life span

Published:2002-05-01 Issue:9 Volume:109 Page:1183-1191
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Jin Hee Kyung,Carter Janet E.,Huntley George W.,Schuchman Edward H.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference30 articles.

1. Acid sphingomyelinase deficient mice: a model of types A and B Niemann–Pick disease

2. Acid sphingomyelinase-deficient mice mimic the neurovisceral form of human lysosomal storage disease (Niemann-Pick disease)

3. BIOCHEMICAL, PATHOLOGICAL, AND CLINICAL RESPONSE TO TRANSPLANTATION OF NORMAL BONE MARROW CELLS INTO ACID SPHINGOMYELINASE-DEFICIENT MICE1

4. Hematopoietic stem cell gene therapy leads to marked visceral organ improvements and a delayed onset of neurological abnormalities in the acid sphingomyelinase deficient mouse model of Niemann–Pick disease

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