Mechanisms by which common variants in the TCF7L2 gene increase risk of type 2 diabetes-Reference-Cited by-同舟云学术

Mechanisms by which common variants in the TCF7L2 gene increase risk of type 2 diabetes

Published:2007-08-01 Issue:8 Volume:117 Page:2155-2163
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Lyssenko Valeriya,Lupi Roberto,Marchetti Piero,Del Guerra Silvia,Orho-Melander Marju,Almgren Peter,Sjögren Marketa,Ling Charlotte,Eriksson Karl-Fredrik,Lethagen υsa-Linda,Mancarella Rita,Berglund Göran,Tuomi Tiinamaija,Nilsson Peter,Del Prato Stefano,Groop Leif

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference45 articles.

1. Large-Scale Association Studies of Variants in Genes Encoding the Pancreatic β-Cell KATP Channel Subunits Kir6.2 (KCNJ11) and SUR1 (ABCC8) Confirm That the KCNJ11 E23K Variant Is Associated With Type 2 Diabetes

2. The common PPARγ Pro12Ala polymorphism is associated with decreased risk of type 2 diabetes

3. Haplotype Structure and Genotype-Phenotype Correlations of the Sulfonylurea Receptor and the Islet ATP-Sensitive Potassium Channel Gene Region

4. Variant of transcription factor 7-like 2 (TCF7L2) gene confers risk of type 2 diabetes

5. Association Analysis of 6,736 U.K. Subjects Provides Replication and Confirms TCF7L2 as a Type 2 Diabetes Susceptibility Gene With a Substantial Effect on Individual Risk

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