Mitochondrial dysfunction in macrophages promotes inflammation and suppresses repair after myocardial infarction
Author:
Funder
NIH
American Heart Association
the Japan Heart Foundation and the Uehara Memorial Foundation
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Link
https://www.jci.org/articles/view/159498/files/pdf
Reference44 articles.
1. Mapping macrophage polarization over the myocardial infarction time continuum
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3. Macrophage polarization: tumor-associated macrophages as a paradigm for polarized M2 mononuclear phagocytes
4. The inflammasome promotes adverse cardiac remodeling following acute myocardial infarction in the mouse
5. IL-10 improves cardiac remodeling after myocardial infarction by stimulating M2 macrophage polarization and fibroblast activation
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