Therapeutic targeting of metabolic vulnerabilities in cancers with MLL3/4-COMPASS epigenetic regulator mutations-Reference-Cited by-同舟云学术

Therapeutic targeting of metabolic vulnerabilities in cancers with MLL3/4-COMPASS epigenetic regulator mutations

Published:2023-07-03 Issue:13 Volume:133 Page:
ISSN:1558-8238
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:

Author:

Zhao Zibo^ORCID,Cao Kaixiang^ORCID,Watanabe Jun,Philips Cassandra N.^ORCID,Zeidner Jacob M.^ORCID,Ishi Yukitomo^ORCID,Wang Qixuan,Gold Sarah R.^ORCID,Junkins Katherine^ORCID,Bartom Elizabeth T.^ORCID,Yue Feng,Chandel Navdeep S.^ORCID,Hashizume Rintaro^ORCID,Ben-Sahra Issam^ORCID,Shilatifard Ali

Funder

National Cancer Institute

EUNICE KENNEDY SHRIVER NATIONAL INSTITUTE OF CHILD HEALTH & HUMAN DEVELOPMENT

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Link

https://www.jci.org/articles/view/169993/files/pdf

Reference70 articles.

1. The MLL3/MLL4 Branches of the COMPASS Family Function as Major Histone H3K4 Monomethylases at Enhancers

2. Enhancer-associated H3K4 monomethylation by Trithorax-related, the Drosophila homolog of mammalian Mll3/Mll4

3. SET1A/COMPASS and shadow enhancers in the regulation of homeotic gene expression

4. A small UTX stabilization domain of Trr is conserved within mammalian MLL3-4/COMPASS and is sufficient to rescue loss of viability in null animals

5. UTX and MLL4 Coordinately Regulate Transcriptional Programs for Cell Proliferation and Invasiveness in Breast Cancer Cells

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