Receptor-Mediated Calcium Entry Is Required for Maximal Effects of Platelet Activating Factor Primed Responses in Human Neutrophils
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference12 articles.
1. Neutrophils, host defense, and inflammation: a double-edged sword
2. Leukocyte-endothelial adhesion molecules
3. Assembly of the phagocyte NADPH oxidase: molecular interaction of oxidase proteins
4. Platelet-activating factor primes neutrophil responses to agonists: role in promoting neutrophil-mediated endothelial damage
5. Neutrophil-activating peptide-1/interleukin 8, a novel cytokine that activates neutrophils.
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1. Calcium influx blocked by SK&F 96365 modulates the LPS plus IFN-γ-induced inflammatory response in murine peritoneal macrophages;International Immunopharmacology;2012-02
2. 2-Aminoethoxydiphenyl borate (2-APB) reduces respiratory burst, MMP-9 release and CD11b expression, and increases l-selectin shedding in bovine neutrophils;Research in Veterinary Science;2012-02
3. Store-operated Ca2+Entry (SOCE) Plays a Role in the Polarization of Neutrophil-like HL-60 Cells by Regulating the Activation of Akt, Src, and Rho Family GTPases;Cellular Physiology and Biochemistry;2012
4. The pro-inflammatory effects of platelet contamination in plasma and mitigation strategies for avoidance;Vox Sanguinis;2011-11-18
5. The anti-inflammatory effect of the SOCC blocker SK&F 96365 on mouse lymphocytes after stimulation by Con A or PMA/ionomycin;Immunobiology;2011-09
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