Mice Disrupted for the KvLQT1 Potassium Channel Regulator IsK Gene Accumulate Mature T Cells
Author:
Publisher
Elsevier BV
Subject
Immunology
Reference28 articles.
1. K(V)LQT1 and IsK (minK) proteins associate to form the I(Ks) cardiac potassium current;Barhanin;Nature,1996
2. Coassembly of K(V)LQT1 and minK (IsK) proteins to form cardiac I(Ks) potassium channel;Sanguinetti;Nature,1996
3. Mutations in Hmink gene cause long QT syndrome and supress IKs function;Splawski;Nature Genet.,1997
4. Positional cloning of a novel potassium channel gene: KVLQT1 mutations cause cardiac arrhythmias;Wang;Nature Genet.,1996
5. Differential expression of KvLQT1 and its regulator IsK in mouse epithelia;Demolombe;Am. J. Physiol. Cell Physiol.,2001
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1. A novel role of the antitumor agent tricyclodecan-9-yl-xanthogenate as an open channel blocker of KCNQ1/KCNE1;European Journal of Pharmacology;2018-04
2. Regulation of Ion Channels, Cellular Carriers and Na(+)/K(+)/ATPase by Janus Kinase 3;Current Medicinal Chemistry;2017-08-23
3. Regulation of Voltage Gated K+ Channel KCNE1/KCNQ1 by the Janus Kinase JAK3;Cellular Physiology and Biochemistry;2015
4. Phenotypic Analysis of Vertigo 2 Jackson Mice with a Kcnq1 Potassium Channel Mutation;Experimental Animals;2007
5. Human congenital long QT syndrome: more than previously thought?;Trends in Pharmacological Sciences;2002-06-01
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