Activation of JNK and Transcriptional Repressor ATF3/LRF1 through the IRE1/TRAF2 Pathway Is Implicated in Human Vascular Endothelial Cell Death by Homocysteine-Reference-Cited by-同舟云学术

Activation of JNK and Transcriptional Repressor ATF3/LRF1 through the IRE1/TRAF2 Pathway Is Implicated in Human Vascular Endothelial Cell Death by Homocysteine

Published:2001-12 Issue:3 Volume:289 Page:718-724
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Zhang Chun,Kawauchi Junya,Adachi Mimi T.,Hashimoto Yoshinori,Oshiro Satoru,Aso Teijiro,Kitajima Shigetaka

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference33 articles.

1. The Metabolic Basis of Inherited Disease;Mudd,1995

2. Homocysteine, a risk factor for premature vascular disease and thrombosis, induces tissue factor activity in endothelial cells.

3. Activation of endogenous factor V by a homocysteine-induced vascular endothelial cell activator.

4. Homocysteine, a thrombogenic agent, suppresses anticoagulant heparan sulfate expression in cultured porcine aortic endothelial cells.

5. Effect of sulfinpyrazone on homocysteine-induced endothelial injury and arteriosclerosis in baboons.

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