PKC ζ Enhances Insulin-like Growth Factor 1-Dependent Mitogenic Activity in the Rat Clonal β Cell Line RIN 1046-38-Reference-Cited by-同舟云学术

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PKC ζ Enhances Insulin-like Growth Factor 1-Dependent Mitogenic Activity in the Rat Clonal β Cell Line RIN 1046-38

Published:2002-01 Issue:1 Volume:290 Page:85-90
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Hennige Anita M.,Fritsche Andreas,Strack Volker,Weigert Cora,Mischak Harald,Borboni Patricia,Renn Walter,Häring Hans-Ulrich,Kellerer Monika

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference28 articles.

1. Protein kinases, protein phosphorylation, and the regulation of insulin secretion from pancreatic beta-cells;Jones;Endocr. Rev.,1998

2. Glucose stimulates islet beta-cell mitogenesis through GTP-binding proteins and by protein kinase C-dependent mechanisms;Sjoholm;Diabetes,1997

3. Glucose-induced translocation of protein kinase C in rat pancreatic islets;Ganesan;Proc. Natl. Acad. Sci. USA,1990

4. Intracellular signaling by hydrolysis of phospholipids and activation of protein kinase C;Nishizuka;Science,1992

5. Atypical protein kinase C isozyme zeta mediates carbachol-stimulated insulin secretion in RINm5F cells;Tang;Diabetes,1998

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1. IGF-1 and Insulin-Receptor Signalling in Insulin-Secreting Cells: From Function to Survival;Islets of Langerhans;2014-02-14

2. IGF-1 and Insulin Receptor Signalling in Insulin-Secreting Cells: From Function to Survival;Islets of Langerhans, 2. ed.;2013

3. Activation of Protein Kinase C-ζ in Pancreatic β-Cells In Vivo Improves Glucose Tolerance and Induces β-Cell Expansion via mTOR Activation;Diabetes;2011-09-16

4. Src tyrosine kinase regulates angiotensin II-induced protein kinase Cζ activation and proliferation in vascular smooth muscle cells;Peptides;2010-06

5. Maternal obesity accelerates fetal pancreatic β-cell but not α-cell development in sheep: prenatal consequences;American Journal of Physiology-Regulatory, Integrative and Comparative Physiology;2009-09

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