Lack of MSH2 involvement differentiates V(D)J recombination from other non-homologous end joining events-Reference-Cited by-同舟云学术

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Lack of MSH2 involvement differentiates V(D)J recombination from other non-homologous end joining events

Published:2005-11-27 Issue:21 Volume:33 Page:6733-6742
ISSN:0305-1048
Container-title:Nucleic Acids Research
language:en
Short-container-title:Nucleic Acids Research

Author:

Larijani M.

Publisher

Oxford University Press (OUP)

Subject

Genetics

Link

http://academic.oup.com/nar/article-pdf/33/21/6733/7125752/gki983.pdf

Cited by 9 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Ataxia Telangiectasia Mutated and MSH2 Control Blunt DNA End Joining in Ig Class Switch Recombination;The Journal of Immunology;2023-01-04

2. Twelve exonic variants in the SLC12A1 and CLCNKB genes alter RNA splicing in a minigene assay;Frontiers in Genetics;2022-08-25

3. The H2B deubiquitinase Usp22 promotes antibody class switch recombination by facilitating non-homologous end joining;Nature Communications;2018-03-08

4. Gata3 restrains B cell proliferation and cooperates with p18INK4c to repress B cell lymphomagenesis;Oncotarget;2016-08-31

5. DNA mismatch repair and the DNA damage response to ionizing radiation: Making sense of apparently conflicting data;Cancer Treatment Reviews;2010-11

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