The H2B deubiquitinase Usp22 promotes antibody class switch recombination by facilitating non-homologous end joining-Reference-Cited by-同舟云学术

The H2B deubiquitinase Usp22 promotes antibody class switch recombination by facilitating non-homologous end joining

Published:2018-03-08 Issue:1 Volume:9 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Li Conglei,Irrazabal Thergiory,So Clare C.,Berru Maribel,Du Likun,Lam Evelyn,Ling Alexanda K.,Gommerman Jennifer L.,Pan-Hammarström Qiang^ORCID,Martin Alberto

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/s41467-018-03455-x.pdf

Reference59 articles.

1. Schatz, D. G. & Ji, Y. Recombination centres and the orchestration of V(D)J recombination. Nat. Rev. Immunol. 11, 251–263 (2011).

2. Maizels, N. Immunoglobulin gene diversification. Annu. Rev. Genet. 39, 23–46 (2005).

3. Casellas, R. et al. Mutations, kataegis and translocations in B cells: understanding AID promiscuous activity. Nat. Rev. Immunol. 16, 164–176 (2016).

4. Muramatsu, M. et al. Class switch recombination and hypermutation require activation-induced cytidine deaminase (AID), a potential RNA editing enzyme. Cell 102, 553–563 (2000).

5. Rush, J. S., Fugmann, S. D. & Schatz, D. G. Staggered AID-dependent DNA double strand breaks are the predominant DNA lesions targeted to S mu in Ig class switch recombination. Int. Immunol. 16, 549–557 (2004).

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