Variants in ampD and dacB lead to in vivo resistance evolution of Pseudomonas aeruginosa within the central nervous system
Author:
Affiliation:
1. Division of Infectious Diseases, Department of Internal Medicine, University of Michigan, SPC 5680, 1150 W. Medical Center Dr., 48109-5680, Ann Arbor, MI, USA
Funder
National Institutes of Health
NIAID
German Research Foundation
Publisher
Oxford University Press (OUP)
Subject
Infectious Diseases,Pharmacology (medical),Pharmacology,Microbiology (medical)
Link
http://academic.oup.com/jac/article-pdf/75/11/3405/33893305/dkaa324.pdf
Reference11 articles.
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2. Pseudomonas aeruginosa Psl is a galactose- and mannose-rich exopolysaccharide;Ma;J Bacteriol,2007
3. Differential β-lactam resistance response driven by ampD or dacB (PBP4) inactivation in genetically diverse Pseudomonas aeruginosa strains;Zamorano;J Antimicrob Chemother,2010
4. Molecular mechanisms of β-lactam resistance mediated by AmpC hyperproduction in Pseudomonas aeruginosa clinical strains;Juan;Antimicrob Agents Chemother,2005
5. Molecular epidemiology of mutations in antimicrobial resistance loci of Pseudomonas aeruginosa isolates from airways of cystic fibrosis patients;Greipel;Antimicrob Agents Chemother,2016
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