Inhibiting the ubiquitin–proteasome system leads to preferential accumulation of toxic N-terminal mutant huntingtin fragments-Reference-Cited by-同舟云学术

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Inhibiting the ubiquitin–proteasome system leads to preferential accumulation of toxic N-terminal mutant huntingtin fragments

Published:2010-03-30 Issue:12 Volume:19 Page:2445-2455
ISSN:1460-2083
Container-title:Human Molecular Genetics
language:en
Short-container-title:

Author:

Li Xiang,Wang Chuan-En,Huang Shanshan,Xu Xingshun,Li Xiao-Jiang,Li He,Li Shihua

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Reference50 articles.

1. Intracellular degradation of misfolded proteins in polyglutamine neurodegenerative diseases;Li;Brain Res. Rev.,2008

2. Rodent genetic models of Huntington disease;Heng;Neurobiol. Dis.,2008

3. N-terminal proteolysis of full-length mutant huntingtin in an inducible PC12 cell model of Huntington's disease;Ratovitski;Cell Cycle,2007

4. Proteolysis of mutant huntingtin produces an exon 1 fragment that accumulates as an aggregated protein in neuronal nuclei in Huntington's disease;Landles;J. Biol. Chem.

5. Aggregation of huntingtin in neuronal intranuclear inclusions and dystrophic neurites in brain;DiFiglia;Science,1997

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