Vitamin B6, B12 and folate modulate deregulated pathways and protein aggregation in yeast model of Huntington disease-Reference-Cited by-同舟云学术

Vitamin B6, B12 and folate modulate deregulated pathways and protein aggregation in yeast model of Huntington disease

Published:2023-02-24 Issue:3 Volume:13 Page:
ISSN:2190-572X
Container-title:3 Biotech
language:en
Short-container-title:3 Biotech

Author:

Pradhan Sai Sanwid^ORCID,Rao K. Raksha^ORCID,Manjunath Meghana^ORCID,Saiswaroop R.^ORCID,Patnana Durga Prasad^ORCID,Phalguna Kanikaram Sai^ORCID,Choudhary Bibha^ORCID,Sivaramakrishnan Venketesh^ORCID

Funder

Department of Biotechnology, Ministry of Science and Technology, India

Department of Science and Technology Science and Engineering Research Board

Department of Biotechnology- Bioinformatics Infrastructure facility

Department of Science and Technology- Fund for improvement of Science and Technology Infrastructure in Higher Educational Institutions

University Grants Commission-Special Assistance Program

The Department of Science & Technology Fund for Improvement of S&T Infrastructure in Higher Educational Institutions

The Department of Biotechnology, India, Glue grant

The Departments Information Technology, Biotechnology and Science and Technology, Government of Karnataka, India

Senior Research Fellowship- Department of Science and Technology‐Innovation in Science Pursuit for Inspired Research, India

Publisher

Springer Science and Business Media LLC

Subject

Agricultural and Biological Sciences (miscellaneous),Environmental Science (miscellaneous),Biotechnology

Link

https://link.springer.com/content/pdf/10.1007/s13205-023-03525-y.pdf

Reference154 articles.

1. Adeyo O, Horn PJ, Lee S et al (2011) The yeast lipin orthologue Pah1p is important for biogenesis of lipid droplets. J Cell Biol 192:1043–1055. https://doi.org/10.1083/jcb.201010111

2. Aditi K, Shakarad MN, Agrawal N (2016) Altered lipid metabolism in Drosophila model of Huntington’s disease. Sci Rep 6:31411. https://doi.org/10.1038/srep31411

3. Alexander AG, Marfil V, Li C (2014) Use of Caenorhabditis elegans as a model to study Alzheimer’s disease and other neurodegenerative diseases. Front Genet. https://doi.org/10.3389/fgene.2014.00279

4. Ames BN, Elson-Schwab I, Silver EA (2002) High-dose vitamin therapy stimulates variant enzymes with decreased coenzyme binding affinity (increased Km): relevance to genetic disease and polymorphisms. Am J Clin Nutr 75:616–658. https://doi.org/10.1093/ajcn/75.4.616

5. Anders S, Huber W (2010) Differential expression analysis for sequence count data. Genome Biol 11:R106. https://doi.org/10.1186/gb-2010-11-10-r106

Cited by 6 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Saccharomyces cerevisiae as a Model for Studying Human Neurodegenerative Disorders: Viral Capsid Protein Expression;International Journal of Molecular Sciences;2023-12-07

2. Cornerstone Cellular Pathways for Metabolic Disorders and Diabetes Mellitus: Non-Coding RNAs, Wnt Signaling, and AMPK;Cells;2023-11-09

3. The impact of aging and oxidative stress in metabolic and nervous system disorders: programmed cell death and molecular signal transduction crosstalk;Frontiers in Immunology;2023-11-08

4. Metabolic deregulation associated with aging modulates protein aggregation in the yeast model of Huntington’s disease;Journal of Biomolecular Structure and Dynamics;2023-09-21

5. The Metabolic Basis for Nervous System Dysfunction in Alzheimer’s Disease, Parkinson’s Disease, and Huntington’s Disease;Current Neurovascular Research;2023-08