JNK1/c-fos inhibits cardiomyocyte TNF- expression via a negative crosstalk with ERK and p38 MAPK in endotoxaemia
Author:
Publisher
Oxford University Press (OUP)
Subject
Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology
Link
http://academic.oup.com/cardiovascres/article-pdf/81/4/733/2160283/cvn336.pdf
Reference36 articles.
1. Inhibition of p38 MAPK decreases myocardial TNF-alpha expression and improves myocardial function and survival in endotoxemia
2. Endothelial Nitric-oxide Synthase Enhances Lipopolysaccharide-stimulated Tumor Necrosis Factor-α Expression via cAMP-mediated p38 MAPK Pathway in Cardiomyocytes
3. Endotoxin-Induced Myocardial Tumor Necrosis Factor-α Synthesis Depresses Contractility of Isolated Rat Hearts
4. Escherichia coli LPS-induced LV dysfunction: role of toll-like receptor-4 in the adult heart
5. Endotoxin stress-response in cardiomyocytes: NF-κB activation and tumor necrosis factor-α expression
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