Tetrahydrocurcumin improves lipopolysaccharide-induced myocardial dysfunction by inhibiting oxidative stress and inflammation via JNK/ERK signaling pathway regulation

Author:

Zhu Hanzhao,Zhang Liyun,Jia Hao,Xu Lu,Cao Yu,Zhai Mengen,Li Kaifeng,Xia Lin,Jiang LiqingORCID,Li Xiang,Zhou Yenong,Liu Jincheng,Yu Shiqiang,Duan WeixunORCID

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Complementary and alternative medicine,Drug Discovery,Pharmaceutical Science,Pharmacology,Molecular Medicine

Reference44 articles.

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2. Organ dysfunction in sepsis–An ominous trajectory from infection to death;Caraballo;Yale J. Biol. Med.,2019

3. Tetrahydrocurcumin, a major metabolite of curcumin, ameliorates allergic airway inflammation by attenuating Th2 response and suppressing the IL-4Ralpha-Jak1-STAT6 and Jagged1/Jagged2 -Notch1/Notch2 pathways in asthmatic mice;Chen;Clin. Exp. Allergy,2018

4. An overview of the non-canonical inflammasome;Downs;Mol. Aspects Med.,2020

5. Assessment of global incidence and mortality of hospital-treated sepsis. Current estimates and limitations;Fleischmann;Am. J. Respir. Crit. Care Med.,2016

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