Arylamine N-acetyltransferase 1 gene regulation by androgens requires a conserved heat shock element for heat shock factor-1
Author:
Publisher
Oxford University Press (OUP)
Subject
Cancer Research,General Medicine
Link
http://academic.oup.com/carcin/article-pdf/31/5/820/7519884/bgq042.pdf
Reference51 articles.
1. Metabolic activation and deactivation of arylamine carcinogens by recombinant human NAT1 and polymorphic NAT2 acetyltransferases
2. N- and O-acetylation of aromatic and heterocyclic amine carcinogens by human monomorphic and polymorphic acetyltransferases expressed in COS-1 cells
3. Pharmacogenetics of the arylamine N-acetyltransferases
4. Polymorphisms of Human N-Acetyltransferases and Cancer Risk
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1. Arylamine N-Acetyltransferases;Reference Module in Biomedical Sciences;2024
2. Population variability of rhesus macaque (Macaca mulatta) NAT1 gene for arylamine N-acetyltransferase 1: Functional effects and comparison with human;Scientific Reports;2019-07-29
3. Genetic and small molecule inhibition of arylamine N -acetyltransferase 1 reduces anchorage-independent growth in human breast cancer cell line MDA-MB-231;Molecular Carcinogenesis;2018-02-03
4. High N-Acetyltransferase 1 Expression is Associated with Estrogen Receptor Expression in Breast Tumors, but is not Under Direct Regulation by Estradiol, 5α-androstane-3β, 17β-Diol, or Dihydrotestosterone in Breast Cancer Cells;Journal of Pharmacology and Experimental Therapeutics;2018-01-16
5. Congenic rats with higher arylamine N-acetyltransferase 2 activity exhibit greater carcinogen-induced mammary tumor susceptibility independent of carcinogen metabolism;BMC Cancer;2017-03-31
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