Genetic and small molecule inhibition of arylamine N -acetyltransferase 1 reduces anchorage-independent growth in human breast cancer cell line MDA-MB-231-Reference-Cited by-同舟云学术

Genetic and small molecule inhibition of arylamine N -acetyltransferase 1 reduces anchorage-independent growth in human breast cancer cell line MDA-MB-231

Published:2018-02-03 Issue:4 Volume:57 Page:549-558
ISSN:0899-1987
Container-title:Molecular Carcinogenesis
language:en
Short-container-title:Mol Carcinog

Author:

Stepp Marcus W.¹,Doll Mark A.¹,Carlisle Samantha M.¹^ORCID,States J. Christopher¹,Hein David W.¹^ORCID

Affiliation:

1. Department of Pharmacology and Toxicology and James Graham Brown Cancer Center; University of Louisville; Louisville, Kentucky

Funder

National Institute of Environmental Health Sciences

Publisher

Wiley

Subject

Cancer Research,Molecular Biology

Reference35 articles.

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2. Folate-dependent hydrolysis of acetyl-coenzyme A by recombinant human and rodent arylamine N-acetyltransferases;Stepp;Biochem Biophys rep,2015

3. Arylamine N-Acetyltransferase-1 is highly expressed in Breast cancers and conveys enhanced growth and resistance to etoposide in vitro;Adam;Mol Cancer Res,2003

4. Gene expression signature of estrogen receptor alpha status in breast cancer;Abba;BMC Genomics,2005

5. Immunohistochemical determination of the miR-1290 target arylamine N-acetyltransferase 1 (NAT1) as a prognostic biomarker in breast cancer;Endo;BMC Cancer,2014

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