Small Molecule Inhibitors of Arylamine N-Acetyltransferase 1 Attenuate Cellular Respiration-Reference-Cited by-同舟云学术

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Small Molecule Inhibitors of Arylamine N-Acetyltransferase 1 Attenuate Cellular Respiration

Published:2024-07-17 Issue:8 Volume:7 Page:2326-2332
ISSN:2575-9108
Container-title:ACS Pharmacology & Translational Science
language:en
Short-container-title:ACS Pharmacol. Transl. Sci.

Author:

Choudhury Chandra¹^ORCID,Egleton James E.²,Butcher Neville J.¹,Russell Angela J.²³^ORCID,Minchin Rodney F.¹^ORCID

Affiliation:

1. School of Biomedical Sciences, The University of Queensland, St Lucia, Brisbane, 4069 Queensland Australia

2. Department of Chemistry, University of Oxford, 12A Mansfield Road, Oxford OX1 3TA, U.K.

3. Department of Pharmacology, University of Oxford, Mansfield Road, OX1 3QT Oxford, U.K.

Funder

National Health and Medical Research Council

Australian Government

Cancer Research UK

Publisher

American Chemical Society (ACS)

Link

https://pubs.acs.org/doi/pdf/10.1021/acsptsci.4c00282

Reference37 articles.

1. RNAi-Mediated Knock-Down of Arylamine N-acetyltransferase-1 Expression Induces E-cadherin Up-Regulation and Cell-Cell Contact Growth Inhibition

2. Small molecule inhibition of arylamine N-acetyltransferase Type I inhibits proliferation and invasiveness of MDA-MB-231 breast cancer cells

3. Knockout of human arylamine N ‐acetyltransferase 1 (NAT1) in MDA‐MB‐231 breast cancer cells leads to increased reserve capacity, maximum mitochondrial capacity, and glycolytic reserve capacity

4. Loss of human arylamine N-acetyltransferase I regulates mitochondrial function by inhibition of the pyruvate dehydrogenase complex

5. Arylamine N-acetyltransferase 1 protects against reactive oxygen species during glucose starvation: Role in the regulation of p53 stability

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