Reconsidering the role of the IL-23/IL-17 immune axis in idiopathic nephrotic syndrome pathogenesis
Author:
Affiliation:
1. Oncology Institute of Southern Switzerland , Bellinzona , Switzerland
Publisher
Oxford University Press (OUP)
Subject
Transplantation,Nephrology
Link
https://academic.oup.com/ckj/advance-article-pdf/doi/10.1093/ckj/sfad264/52772764/sfad264.pdf
Reference9 articles.
1. Focal segmental glomerulosclerosis associated with the use of the IL-23 inhibitor guselkumab;Stryckers;Clin Kidney J,2023
2. Current understanding of the molecular mechanisms of circulating permeability factor in focal segmental glomerulosclerosis;Salfi;Front Immunol,2023
3. Human Th17 cells produce a soluble mediator that increases podocyte motility via signaling pathways that mimic PAR-1 activation;May;Am J Physiol Renal Physiol,2019
4. Th17/Treg imbalance in adult patients with minimal change nephrotic syndrome;Liu;Clin Immunol,2011
5. The role of Th17/IL-17 in the pathogenesis of primary nephrotic syndrome in children;Wang;Kidney Blood Press Res,2013
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