A transcriptional serenAID: the role of noncoding RNAs in class switch recombination
Author:
Affiliation:
1. Immunology Program, Memorial Sloan Kettering Cancer, New York, NY 10065, USA and
2. Immunology and Microbial Pathogenesis Program, Weill Cornell Graduate School of Medical Sciences, New York, NY 10065, USA
Funder
National Institutes of Health
Publisher
Oxford University Press (OUP)
Subject
Immunology,General Medicine,Immunology and Allergy
Link
http://academic.oup.com/intimm/article-pdf/29/4/183/24325263/dxx027.pdf
Reference121 articles.
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2. Class switch recombination and hypermutation require activation-induced cytidine deaminase (AID), a potential RNA editing enzyme;Muramatsu;Cell,2000
3. Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the Hyper-IgM syndrome (HIGM2);Revy;Cell,2000
4. Mismatch recognition and uracil excision provide complementary paths to both Ig switching and the A/T-focused phase of somatic mutation;Rada;Mol. Cell,2004
5. AID mutates E. coli suggesting a DNA deamination mechanism for antibody diversification;Petersen-Mahrt;Nature,2002
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