The Neuroinflammatory Phenotype in a Mouse Model of Gulf War Illness is Unrelated to Brain Regional Levels of Acetylcholine as Measured by Quantitative HILIC-UPLC-MS/MS
Author:
Affiliation:
1. Health Effects Laboratory Division
2. Respiratory Health Division, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, Morgantown, West Virginia 26505
Funder
Congressionally Directed Medical Research Programs
Gulf War Illness Research Program
Centers for Disease Control and Prevention
National Institute for Occupational Safety and Health
Publisher
Oxford University Press (OUP)
Subject
Toxicology
Link
http://academic.oup.com/toxsci/article-pdf/165/2/302/25818642/kfy130.pdf
Reference58 articles.
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2. Brain fixation for acetylcholine measurements;Bertrand;J. Neurosci. Methods,1994
3. Biogenesis of acetylcholinesterase is impaired, although its mRNA level remains normal, in the glucocorticoid-treated rat skeletal muscle;Brank;Eur. J. Biochem.,1998
4. Behavioral and neurochemical effects of acute chlorpyrifos in rats: Tolerance to prolonged inhibition of cholinesterase;Bushnell;J. Pharmacol. Exp. Ther.,1993
5. The Role of Target Site Activation of Phosphorothionates in Acute Toxicity
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