Enhanced inactivation and pH sensitivity of Na+ channel mutations causing hypokalaemic periodic paralysis type II
Author:
Publisher
Oxford University Press (OUP)
Subject
Neurology (clinical)
Link
http://academic.oup.com/brain/article-pdf/125/4/835/17864569/1250835.pdf
Reference33 articles.
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2. Bulman DE, Scoggan KA, van Oene MD, Nicolle MW, Hahn AF, Tollar LL, et al. A novel sodium channel mutation in a family with hypokalemic periodic paralysis. Neurology1999; 53: 1932–6.
3. Cannon SC, Brown RH, Corey DP. A sodium channel defect in hyperkalemic periodic paralysis: potassium‐induced failure of inactivation. Neuron1991; 6: 619–26.
4. Cannon SC, Brown RH, Corey DP. Theoretical reconstruction of myotonia and paralysis caused by incomplete inactivation of sodium channels. Biophys J1993; 65: 270–88.
5. Cha A, Ruben PC, George AL Jr, Fujimoto E, Bezanilla F. Voltage sensors in domains III and IV, but not I and II, are immobilized by Na+ channel fast inactivation. Neuron1999; 22: 73–87.
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