Autoimmune ‘secondary synaptopathies’: do NMDAR antibodies cause a primary extra-synaptopathy?-Reference-Cited by-同舟云学术

Autoimmune ‘secondary synaptopathies’: do NMDAR antibodies cause a primary extra-synaptopathy?

Published:2024-07-29 Issue:8 Volume:147 Page:2601-2603
ISSN:0006-8950
Container-title:Brain
language:en
Short-container-title:

Author:

Zhao Meng¹²,Lynch David R³,Irani Sarosh R¹²⁴⁵^ORCID

Affiliation:

1. Department of Neurology, Mayo Clinic , Jacksonville, FL 32224 , USA

2. Department of Neuroscience, Mayo Clinic , Jacksonville, FL 32224 , USA

3. Department of Neurology, University of Pennsylvania School of Medicine , Philadelphia, PA 19104 , USA

4. Nuffield Department of Clinical Neurosciences, University of Oxford , Oxford OX3 9DU , UK

5. Department of Neurology, John Radcliffe Hospital, Oxford University Hospitals , Oxford OX3 9DU , UK

Abstract

This scientific commentary refers to ‘NMDA receptor autoantibodies primarily impair the extrasynaptic compartment’ by Jamet et al. (https://doi.org/10.1093/brain/awae163).

Funder

Medical Research Council

Wellcome Trust Fellowship

National Institute for Health Research

Oxford Biomedical Research Centre

Publisher

Oxford University Press (OUP)

Link

https://academic.oup.com/brain/advance-article-pdf/doi/10.1093/brain/awae236/58677050/awae236.pdf

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