Short antisense-locked nucleic acids (all-LNAs) correct alternative splicing abnormalities in myotonic dystrophy-Reference-Cited by-同舟云学术

Short antisense-locked nucleic acids (all-LNAs) correct alternative splicing abnormalities in myotonic dystrophy

Published:2015-03-09 Issue:6 Volume:43 Page:3318-3331
ISSN:1362-4962
Container-title:Nucleic Acids Research
language:en
Short-container-title:

Author:

Wojtkowiak-Szlachcic Agnieszka,Taylor Katarzyna,Stepniak-Konieczna Ewa,Sznajder Lukasz J.,Mykowska Agnieszka,Sroka Joanna,Thornton Charles A.,Sobczak Krzysztof

Publisher

Oxford University Press (OUP)

Subject

Genetics

Link

http://academic.oup.com/nar/article-pdf/43/6/3318/17434924/gkv163.pdf

Reference56 articles.

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3. Expansion of a CUG trinucleotide repeat in the 3′ untranslated region of myotonic dystrophy protein kinase transcripts results in nuclear retention of transcripts;Davis;Proc. Natl. Acad. Sci. U.S.A.,1997

4. Recruitment of human muscleblind proteins to (CUG)(n) expansions associated with myotonic dystrophy;Miller;EMBO J.,2000

5. Transcriptome-wide regulation of pre-mRNA splicing and mRNA localization by muscleblind proteins;Wang;Cell,2012

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