Global hypermethylation in fetal cortex of Down syndrome due to DNMT3L overexpression-Reference-Cited by-同舟云学术

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Global hypermethylation in fetal cortex of Down syndrome due to DNMT3L overexpression

Published:2016-02-23 Issue:9 Volume:25 Page:1714-1727
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:Hum. Mol. Genet.

Author:

Lu Jie,Mccarter Monika,Lian Gewei,Esposito Giuseppe,Capoccia Elena,Delli-Bovi Laurent C.,Hecht Jonathan,Sheen Volney

Funder

National Institute of Neurological Disorders and Stroke

Roswell Park Cancer Institute and National Cancer Institute

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/25/9/1714/18527970/ddw043.pdf

Reference52 articles.

1. Lu J. , Delli-Bovi L.C. , Hecht J. , Folkerth R. , Sheen V.L. (2011) Generation of neural stem cells from discarded human fetal cortical tissue. J. Vis. Exp., in press.

2. Genomic and functional profiling of human Down syndrome neural progenitors implicates S100B and aquaporin 4 in cell injury

3. S100B induces tau protein hyperphosphorylation via Dickopff-1 up-regulation and disrupts the Wnt pathway in human neural stem cells

4. S100B and APP Promote a Gliocentric Shift and Impaired Neurogenesis in Down Syndrome Neural Progenitors

5. OLIG2 over-expression impairs proliferation of human Down syndrome neural progenitors

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