Role of MEL-18 Amplification in Anti-HER2 Therapy of Breast Cancer-Reference-Cited by-同舟云学术

Role of MEL-18 Amplification in Anti-HER2 Therapy of Breast Cancer

Published:2018-09-27 Issue:6 Volume:111 Page:609-619
ISSN:0027-8874
Container-title:JNCI: Journal of the National Cancer Institute
language:en
Short-container-title:

Author:

Lee Jeong-Yeon¹,Joo Hyeong-Seok²,Choi Hee-Joo²,Jin Sora²,Kim Hyung-Yong²,Jeong Ga-Young²,An Hee Woon²,Park Mi Kyung³,Lee Seung Eun⁴,Kim Wan-Seop⁴,Son Taekwon⁵,Min Kyueng-Whan²,Oh Young-Ha²,Kong Gu²

Affiliation:

1. Department of Medicine, College of Medicine

2. Department of Pathology, College of Medicine

3. Hanyang University, Seoul, Republic of Korea; National Cancer Center, Goyang, Republic of Korea

4. Department of Pathology, Konkuk University Medical Center, Konkuk University School of Medicine, Seoul, Republic of Korea

5. Research Institute of Pharmaceutical Sciences, College of Pharmacy, Seoul National University, Seoul, Republic of Korea

Funder

National Research Foundation

Publisher

Oxford University Press (OUP)

Subject

Cancer Research,Oncology

Link

http://academic.oup.com/jnci/article-pdf/111/6/609/28796245/djy151.pdf

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4. Mel-18 negatively regulates INK4a/ARF-independent cell cycle progression via Akt inactivation in breast cancer;Lee;Cancer Res,2008

5. Loss of Mel-18 induces tumor angiogenesis through enhancing the activity and expression of HIF-1alpha mediated by the PTEN/PI3K/Akt pathway;Park;Oncogene,2011

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