TDP-43 loss of cellular function through aggregation requires additional structural determinants beyond its C-terminal Q/N prion-like domain-Reference-Cited by-同舟云学术

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TDP-43 loss of cellular function through aggregation requires additional structural determinants beyond its C-terminal Q/N prion-like domain

Published:2014-08-13 Issue:1 Volume:24 Page:9-20
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:

Author:

Budini Mauricio,Romano Valentina,Quadri Zainuddin,Buratti Emanuele,Baralle Francisco E.

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Reference54 articles.

1. TDP-43: gumming up neurons through protein-protein and protein-RNA interactions;Trends Biochem. Sci.,2012

2. Structural determinants of the cellular localization and shuttling of TDP-43;J. Cell Sci.,2008

3. Characterization and functional implications of the RNA binding properties of nuclear factor TDP-43, a novel splicing regulator of CFTR exon 9;J. Biol. Chem.,2001

4. Nuclear factor TDP-43 binds to the polymorphic TG repeats in CFTR intron 8 and causes skipping of exon 9: a functional link with disease penetrance;Am. J. Hum. Genet.,2004

5. Autoregulation of TDP-43 mRNA levels involves interplay between transcription, splicing, and alternative polyA site selection;Genes Dev.,2012

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