β-glucan suppresses cell death of ASC deficient macrophages invaded by periodontopathic bacteria through the caspase-11 pathway

Author:

Inoue Maki12,Okinaga Toshinori3,Usui Michihiko2,Kawano Aki1,Thongsiri Chuencheewit2,Nakashima Keisuke2,Ariyoshi Wataru1,Nishihara Tatsuji1

Affiliation:

1. Division of Infections and Molecular Biology, Kyushu Dental University, Kitakyushu, Japan

2. Division of Periodontology, Kyushu Dental University, Kitakyushu, Japan

3. Department of Bacteriology, Osaka Dental University, Osaka, Japan

Publisher

Oxford University Press (OUP)

Subject

Genetics,Molecular Biology,Microbiology

Reference34 articles.

1. Pyroptosis: host cell death and inflammation;Bergsbaken;Nat Rev Microbiol,2009

2. Caspase-11 increases susceptibility to Salmonella infection in the absence of caspase-1;Broz;Nature,2012

3. Recognition and modulation of Dectin-1 and TLR2 receptors by curdlan derivatives and purified natural extracts;Calugaru;Roum Arch Microbiol Immunol,2009

4. Caspase-11 activation in response to bacteria secretion systems that access the host cytosol;Casson;PLoS Pathog,2013

5. Pore-forming activity and structural autoinhibition of the gasdermin family;Ding;Nature,2016

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