The response to DNA damage induced by 4-nitroquinoline-1-oxide or its 3-methyl derivative in xeroderma pigmentosum fibroblasts belonging to different complementation groups: evidence for different epistasis groups involved in the repair of large adducts in human DNA
Author:
Publisher
Oxford University Press (OUP)
Subject
Cancer Research,General Medicine
Link
http://academic.oup.com/carcin/article-pdf/8/8/1071/7521627/8-8-1071.pdf
Cited by 21 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. GCN5-deficiency remarkably enhances the sensitivity of B cells in response to 4-nitroquinoline 1-oxide;Fundamental Toxicological Sciences;2016
2. Inverse correlation between p53 protein levels and DNA repair efficiency in human fibroblast strains treated with 4-nitroquinoline 1-oxide: evidence that lesions other than DNA strand breaks trigger the p53 response;Carcinogenesis;1999-06-01
3. Faculty DNA polymerase δ/ɛ-mediated excision repair in response to γ radiation or ultraviolet light in p53-deficient fibroblast strains from affected members of a cancer-prone family with Li-Fraumeni syndrome;Carcinogenesis;1996
4. Effect of DNA polymerase inhibitors on repair of γ ray-induced DNA damage in proliferating (intact versus permeable) human fibroblasts: evidence for differences in the modes of action of aphidicolin and 1-β-d-arabinofuranosylcytosine;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;1994-10
5. Hyperresistance to 4-nitroquinoline 1-oxide cytotoxicity and reduced DNA damage formation in dermal fibroblast strains derived from five members of a cancer-prone family;British Journal of Cancer;1993-11
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