GCN5-deficiency remarkably enhances the sensitivity of B cells in response to 4-nitroquinoline 1-oxide-Reference-Cited by-同舟云学术

GCN5-deficiency remarkably enhances the sensitivity of B cells in response to 4-nitroquinoline 1-oxide

Published:2016 Issue:3 Volume:3 Page:137-142
ISSN:2189-115X
Container-title:Fundamental Toxicological Sciences
language:en
Short-container-title:Fundam. Toxicol. Sci.

Author:

Kikuchi Hidehiko¹²,Kuribayashi Futoshi²³,Mimuro Hitomi²,Imajoh-Ohmi Shinobu⁴

Affiliation:

1. Laboratory of Biological Chemistry, Department of Food and Nutrition, Shokei University Junior College

2. Division of Bacteriology, Department of Infectious Diseases Control, International Research Center for Infectious Diseases, The Institute of Medical Science, The University of Tokyo

3. Department of Biochemistry, Kawasaki Medical School

4. Laboratory Center for Proteomics Research, Graduate School of Frontier Sciences, The Institute of Medical Science, The University of Tokyo

Publisher

Japanese Society of Toxicology

Link

https://www.jstage.jst.go.jp/article/fts/3/3/3_137/_pdf

Reference41 articles.

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3. Brand, M., Moggs, J.G., Oulad-Abdelghani, M., Lejeune, F., Dilworth, F.J., Stevenin, J., Almouzni, G. and Tora, L. (2001): UV-damaged DNA-binding protein in the TFTC complex links DNA damage recognition to nucleosome acetylation. EMBO J., 20, 3187-3196.

4. Brownell, J.E., Zhou, J., Ranalli, T., Kobayashi, R., Edomondson, D.G., Roth, S.Y. and Allis, C.D. (1996): Tetrahymena histone acetyltransferase A: a homolog to yeast Gcn5p linking histone acetylation to gene activation. Cell, 84, 843-851.

5. Buerstedde, J.M. and Takeda, S. (1991): Increased ration of targeted to random integration after transfection of chicken B cell lines. Cell, 67, 179-188.

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