Deletion of Gab2 in mice protects against hepatic steatosis and steatohepatitis: a novel therapeutic target for fatty liver disease-Reference-Cited by-同舟云学术

Deletion of Gab2 in mice protects against hepatic steatosis and steatohepatitis: a novel therapeutic target for fatty liver disease

Published:2016-06-09 Issue:6 Volume:8 Page:492-504
ISSN:1674-2788
Container-title:Journal of Molecular Cell Biology
language:en
Short-container-title:J Mol Cell Biol

Author:

Chen Shuai,Kang Yujia,Sun Yan,Zhong Yanhong,Li Yanli,Deng Lijuan,Tao Jin,Li Yang,Tian Yingpu,Zhao Yinan,Cheng Jianghong,Liu Wenjie,Feng Gen-Sheng,Lu Zhongxian

Funder

National Basic Research Program of China

National Natural Science Foundation of China

Publisher

Oxford University Press (OUP)

Subject

Cell Biology,Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/jmcb/article-pdf/8/6/492/18526187/mjw028.pdf

Reference40 articles.

1. GAB2—a Scaffolding Protein in Cancer: Figure 1.

2. Inhibition of JNK along with activation of ERK1/2 MAPK pathways improve steatohepatitis among the rats

3. Deletion of Gab1 in the liver leads to enhanced glucose tolerance and improved hepatic insulin action

4. Concerted Functions of Gab1 and Shp2 in Liver Regeneration and Hepatoprotection

5. A role for the scaffolding adapter GAB2 in breast cancer

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