Cucurbitacin B ameliorates AKT-driven fatty liver disease in mice by suppressing AKT/mTORC1 and LXRα/SREBP1 lipogenic pathways-Reference-Cited by-同舟云学术

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Cucurbitacin B ameliorates AKT-driven fatty liver disease in mice by suppressing AKT/mTORC1 and LXRα/SREBP1 lipogenic pathways

Published:2024-02 Issue: Volume:113 Page:106051
ISSN:1756-4646
Container-title:Journal of Functional Foods
language:en
Short-container-title:Journal of Functional Foods

Author:

Wang Chuting,Sheng Lei,Deng Dongjie,Chen Zhiwei,Chen Xin,Meng Yan,Wang Qi,Wang Guihong,Zheng Guohua,Hu Junjie^ORCID

Funder

Hebei University of Chinese Medicine

Publisher

Elsevier BV

Reference43 articles.

1. Cucurbitacins – A promising target for cancer therapy;Alghasham;International Journal of Health Sciences (Qassim),2013

2. Connecting mTORC1 signaling to SREBP-1 activation;Bakan;Current Opinion in Lipidology,2012

3. Clinical model for NASH and advanced fibrosis in adult patients with diabetes and NAFLD: guidelines for referral in NAFLD;Bazick;Diabetes Care,2015

4. Molecular mediators of hepatic steatosis and liver injury;Browning;The Journal of Clinical Investigation,2004

5. The multiple-hit pathogenesis of non-alcoholic fatty liver disease (NAFLD);Buzzetti;Metabolism,2016

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