Activation of p38 signaling increases utrophin A expression in skeletal muscle via the RNA-binding protein KSRP and inhibition of AU-rich element-mediated mRNA decay: implications for novel DMD therapeutics-Reference-Cited by-同舟云学术

Activation of p38 signaling increases utrophin A expression in skeletal muscle via the RNA-binding protein KSRP and inhibition of AU-rich element-mediated mRNA decay: implications for novel DMD therapeutics

Published:2013-04-10 Issue:15 Volume:22 Page:3093-3111
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:Human Molecular Genetics

Author:

Amirouche A.,Tadesse H.,Lunde J. A.,Belanger G.,Cote J.,Jasmin B. J.

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/22/15/3093/1846326/ddt165.pdf

Reference105 articles.

1. Isolation of candidate cDNAs for portions of the Duchenne muscular dystrophy gene

2. Complete cloning of the duchenne muscular dystrophy (DMD) cDNA and preliminary genomic organization of the DMD gene in normal and affected individuals

3. Chapter 9 The Value of Mammalian Models for Duchenne Muscular Dystrophy in Developing Therapeutic Strategies

4. Molecular, cellular, and pharmacological therapies for Duchenne/Becker muscular dystrophies

5. Dystrophin, its interactions with other proteins, and implications for muscular dystrophy

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