α1AMPK deletion in myelomonocytic cells induces a pro-inflammatory phenotype and enhances angiotensin II-induced vascular dysfunction-Reference-Cited by-同舟云学术

α1AMPK deletion in myelomonocytic cells induces a pro-inflammatory phenotype and enhances angiotensin II-induced vascular dysfunction

Published:2018-07-04 Issue:14 Volume:114 Page:1883-1893
ISSN:0008-6363
Container-title:Cardiovascular Research
language:en
Short-container-title:

Author:

Jansen Thomas¹,Kröller-Schön Swenja¹,Schönfelder Tanja²,Foretz Marc³⁴⁵,Viollet Benoit³⁴⁵,Daiber Andreas¹,Oelze Matthias¹,Brandt Moritz¹²,Steven Sebastian¹²,Kvandová Miroslava¹,Kalinovic Sanela¹,Lagrange Jeremy²,Keaney John F⁶,Münzel Thomas¹,Wenzel Philip¹²,Schulz Eberhard¹

Affiliation:

1. Department of Cardiology 1, Center for Cardiology, Universitätsmedizin Mainz, Mainz, Germany

2. Center for Thrombosis and Hemostasis (CTH), Universitätsmedizin Mainz, Mainz, Germany

3. Institut Cochin, INSERM U1016, Paris, France

4. CNRS, UMR8104, Paris, France

5. Université Paris Descartes, Sorbonne Paris cité, 24 rue du faubourg Saint Jacques, Paris, France

6. Division of Cardiovascular Medicine, UMass Medical School, 55 Lake Avenue North, Worcester, MA, USA

Funder

German Research Foundation

German Federal Ministry for Education and Research

European Commission

Publisher

Oxford University Press (OUP)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://academic.oup.com/cardiovascres/article-pdf/114/14/1883/26968561/cvy172.pdf

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5. Inflammation, immunity, and hypertensive end-organ damage;McMaster;Circ Res,2015

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