Molecular mechanisms of secondary hyperparathyroidism
Author:
Affiliation:
1. Minerva Center for Calcium and Bone Metabolism, Nephrology Services, Hadassah Hospital and Hebrew University Hadassah Medical School, PO Box 12000, Jerusalem 91120, Israel
Publisher
Oxford University Press (OUP)
Subject
Transplantation,Nephrology
Link
http://academic.oup.com/ndt/article-pdf/15/suppl_5/2/36974899/1505s0002.pdf
Reference19 articles.
1. Regulation of 1,25-dihydroxyvitamin D3 receptor gene expression by 1,25-dihydroxyvitamin D3 in the parathyroid in vivo;Naveh-Many;J Clin Invest,1990
2. Regulation by vitamin D metabolites of parathyroid hormone gene transcription in vivo in the rat;Silver;J Clin Invest,1986
3. Decreased 1,25-dihydroxyvitamin D3 receptor density is associated with a more severe form of parathyroid hyperplasia in chronic uremic patients;Fukuda;J Clin Invest,1993
4. Cloning and characterization of an extracellular Ca2+-sensing receptor from bovine parathyroid;Brown;Nature,1993
5. Regulation of parathyroid hormone gene expression by hypocalcemia, hypercalcemia, and vitamin D in the rat;Naveh-Many;J Clin Invest,1990
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