Intracellular localization and splicing regulation of FUS/TLS are variably affected by amyotrophic lateral sclerosis-linked mutations
Author:
Publisher
Oxford University Press (OUP)
Subject
Genetics
Link
http://academic.oup.com/nar/article-pdf/39/7/2781/16781033/gkq1162.pdf
Reference69 articles.
1. Fusion of the dominant negative transcription regulator CHOP with a novel gene FUS by translocation t(12;16) in malignant liposarcoma;Rabbitts;Nat. Genet.,1993
2. An RNA-binding protein gene, TLS/FUS, is fused to ERG in human myeloid leukemia with t(16;21) chromosomal translocation;Ichikawa;Cancer Res.,1994
3. Fusion of CHOP to a novel RNA-binding protein in human myxoid liposarcoma;Crozat;Nature,1993
4. A novel effector domain from the RNA-binding protein TLS or EWS is required for oncogenic transformation by CHOP;Zinszner;Genes Dev.,1994
5. RNA-binding protein TLS is a major nuclear aggregate-interacting protein in huntingtin exon 1 with expanded polyglutamine-expressing cells;Doi;J. Biol. Chem.,2008
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