Cell cycle regulation by FasL and Apo2L/TRAIL in human T-cell blasts. Implications for autoimmune lymphoproliferative syndromes-Reference-Cited by-同舟云学术

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Cell cycle regulation by FasL and Apo2L/TRAIL in human T-cell blasts. Implications for autoimmune lymphoproliferative syndromes

Published:2008-05-15 Issue:2 Volume:84 Page:488-498
ISSN:0741-5400
Container-title:Journal of Leukocyte Biology
language:en
Short-container-title:Journal of Leukocyte Biology

Author:

Bosque Alberto,Aguiló Juan I.,del Rey Manuel,Paz-Artal Estela,Allende Luis M.,Naval Javier,Anel Alberto

Funder

Ministerio de Educación, Gobierno de Chile

Genentech

Universidad de Zaragoza

Publisher

Wiley

Subject

Cell Biology,Immunology,Immunology and Allergy

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1189/jlb.0108043/fullpdf

Reference49 articles.

1. Lymphoproliferation disorder explained by defects in Fas antigen that mediates apoptosis;Watanabe-Fukunaga;Nature,1992

2. Generalized lymphoproliferative disease in mice, caused by a point mutation in the Fas ligand;Takahashi;Cell,1994

3. Dominant interfering Fas gene mutations impair apoptosis in a human autoimmune lymphoproliferative syndrome;Fisher;Cell,1995

4. Mutations in Fas associated with human lymphoproliferative syndrome and autoimmunity;Rieux-Laucat;Science,1995

5. Genetic disorders of programmed cell death in the immune system;Bidère;Annu. Rev. Immunol.,2006

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