Immunologic evaluation and genetic defects of apoptosis in patients with autoimmune lymphoproliferative syndrome (ALPS)-Reference-Cited by-同舟云学术

Immunologic evaluation and genetic defects of apoptosis in patients with autoimmune lymphoproliferative syndrome (ALPS)

Published:2020-12-23 Issue:4 Volume:58 Page:253-274
ISSN:1040-8363
Container-title:Critical Reviews in Clinical Laboratory Sciences
language:en
Short-container-title:Critical Reviews in Clinical Laboratory Sciences

Author:

Casamayor-Polo Laura¹^ORCID,López-Nevado Marta¹^ORCID,Paz-Artal Estela¹²³^ORCID,Anel Alberto⁴^ORCID,Rieux-Laucat Frederic⁵^ORCID,Allende Luis M.¹²³^ORCID

Affiliation:

1. Instituto de Investigación Sanitaria Hospital 12 de Octubre (imas12), Madrid, Spain

2. Immunology Department, University Hospital 12 de Octubre, Madrid, Spain

3. School of Medicine, University Hospital 12 de Octubre, Complutense University of Madrid, Madrid, Spain

4. Apoptosis, Immunity and Cancer Group, University of Zaragoza/Aragón Health Research Institute (IIS-Aragón), Zaragoza, Spain

5. Laboratory of Immunogenetics of Pediatric Autoimmune Diseases, Université de Paris, Imagine Institute, INSERM UMR 1163, Paris, France

Funder

Fondo de Investigación Sanatoria

FEDER

Fondo Social Europeo

Programa Operativo de Empleo Juvenil y la Iniciativa de Empleo Juvenil

Institut National de la Santé et de la Recherche Médicale

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Center de Référence Déficits Immunitaires Héréditaires

Publisher

Informa UK Limited

Subject

Biochemistry, medical,Clinical Biochemistry,General Biochemistry, Genetics and Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/10408363.2020.1855623

Reference136 articles.

1. Apoptosis: A Basic Biological Phenomenon with Wideranging Implications in Tissue Kinetics

2. Regulation of Lymphocyte Survival by the BCL-2 Gene Family

3. Deletion of self-reactive T cells before entry into the thymus medulla

4. Tolerance in T-cell-receptor transgenic mice involves deletion of nonmature CD4+8+ thymocytes

5. Negative selection — clearing out the bad apples from the T-cell repertoire

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