COMPENSATED HEREDITARY HAEMOLYTIC DISEASE RESULTING FROM AN UNSTABLE HAEMOGLOBIN FRACTION
Author:
Affiliation:
1. Hæmatology DepartmentKanematsu Memorial Institute of Pathology, Sydney Hospital
Publisher
AMPCo
Subject
General Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.5694/j.1326-5377.1967.tb21762.x
Reference12 articles.
1. Hemoglobin Zürich. II. Physicochemical Properties of the Abnormal Hemoglobin;Bachman F.;Blood,1962
2. The Hemolytic Effect of Primaquine. IV. An In‐vitro Test for Sensitivity of Erythrocytes to Primaquine;Beutler E.;J. Lab. clin. Med.,1955
3. Haemoglobin Köln (β–98 Valine → Methionine): An Unstable Protein Causing Inclusion-Body Anaemia
4. Hereditary Heinz-Body Anaemia.
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1. Haemoglobin Manukau β67[E11] Val→Gly: transfusion-dependent haemolytic anaemia ameliorated by coexisting alpha thalassaemia;British Journal of Haematology;1993-10
2. Hemoglobin casper: β 106 (G8) Leu→Pro;The American Journal of Medicine;1973-10
3. Introduction to discussion of Heinz body anemias—Unstable hemoglobinopathies;Experimental Eye Research;1971-05
4. Idiopathic Heinz Body Anaemia: Hb-Bristol (β67 (E11) Val→Asp);British Journal of Haematology;1970-04
5. Properties of hemoglobin M, milwaukee-1 variant and its unique characteristic;Biochimica et Biophysica Acta (BBA) - Protein Structure;1969-11
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