Differences in susceptibility to ADR nephropathy among C57BL/6 substrains
Author:
Affiliation:
1. Laboratory of Laboratory Animal Science and Medicine, School of Veterinary Medicine, Kitasato University, 35-1 Higashi-23, Towada, Aomori 034-8628, Japan
Publisher
Japanese Association for Laboratory Animal Science
Subject
General Veterinary,General Biochemistry, Genetics and Molecular Biology,Animal Science and Zoology,General Medicine
Link
https://www.jstage.jst.go.jp/article/expanim/72/4/72_23-0003/_pdf
Reference20 articles.
1. 1. Nogueira A, Pires MJ, Oliveira PA. Pathophysiological mechanisms of renal fibrosis: a review of animal models and therapeutic strategies. In Vivo. 2017; 31: 1–22.
2. 2. Fogo AB. Animal models of FSGS: lessons for pathogenesis and treatment. Semin Nephrol. 2003; 23: 161–171.
3. 3. Lee VW, Harris DC. Adriamycin nephropathy: a model of focal segmental glomerulosclerosis. Nephrology (Carlton). 2011; 16: 30–38.
4. 4. Pettitt SJ, Liang Q, Rairdan XY, Moran JL, Prosser HM, Beier DR, et al. Agouti C57BL/6N embryonic stem cells for mouse genetic resources. Nat Methods. 2009; 6: 493–495.
5. 5. Arif E, Solanki AK, Nihalani D. Adriamycin susceptibility among C57BL/6 substrains. Kidney Int. 2016; 89: 721–723.
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