Adriamycin susceptibility among C57BL/6 substrains
Author:
Publisher
Elsevier BV
Subject
Nephrology
Reference5 articles.
1. Extensive podocyte loss triggers a rapid parietal epithelial cell response;Hakroush;J Am Soc Nephrol,2014
2. Podocyte-specific deletion of Myh9 encoding nonmuscle myosin heavy chain 2A predisposes mice to glomerulopathy;Johnstone;Mol Cell Bi006Fl,2011
3. A comparative phenotypic and genomic analysis of C57BL/6J and C57BL/6N mouse strains;Simon;Genome Biol,2013
4. Adriamycin alters glomerular endothelium to induce proteinuria;Jeansson;J Am Soc Nephrol,2009
5. Podocyte polarity signalling;Simons;Curr Opin Nephrol Hypertens,2009
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2. Differences in susceptibility to ADR nephropathy among C57BL/6 substrains;Experimental Animals;2023
3. Adriamycin-Induced Nephropathy is Robust in N and Modest in J Substrain of C57BL/6;Frontiers in Cell and Developmental Biology;2022-06-16
4. Nuclear Receptor Interacting Protein-2 Mediates the Stabilization and Activation of β-Catenin During Podocyte Injury;Frontiers in Cell and Developmental Biology;2021-12-24
5. Repurposing Riociguat to Target a Novel Paracrine Nitric Oxide-TRPC6 Pathway to Prevent Podocyte Injury;International Journal of Molecular Sciences;2021-11-19
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