Elucidation of Proto-Oncogene C-Abl Function with the Use of Mouse Models and the Disease Model of Chronic Myeloid Leukemia
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Apple Academic Press
Reference160 articles.
1. Targeting PIM Kinases Impairs Survival of Hematopoietic Cells Transformed by Kinase Inhibitor–Sensitive and Kinase Inhibitor–Resistant Forms of Fms-Like Tyrosine Kinase 3 and BCR/ABL
2. Interaction of c-Abl and p73α and their collaboration to induce apoptosis
3. Identification of mcl-1 as a BCR/ABL-dependent target in chronic myeloid leukemia (CML): evidence for cooperative antileukemic effects of imatinib and mcl-1 antisense oligonucleotides
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