Targeting PIM Kinases Impairs Survival of Hematopoietic Cells Transformed by Kinase Inhibitor–Sensitive and Kinase Inhibitor–Resistant Forms of Fms-Like Tyrosine Kinase 3 and BCR/ABL-Reference-Cited by-同舟云学术

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Targeting PIM Kinases Impairs Survival of Hematopoietic Cells Transformed by Kinase Inhibitor–Sensitive and Kinase Inhibitor–Resistant Forms of Fms-Like Tyrosine Kinase 3 and BCR/ABL

Published:2006-04-01 Issue:7 Volume:66 Page:3828-3835
ISSN:0008-5472
Container-title:Cancer Research
language:en
Short-container-title:Cancer Res

Author:

Adam Myriam,Pogacic Vanda,Bendit Marina,Chappuis Richard,Nawijn Martijn C.,Duyster Justus,Fox Casey J.,Thompson Craig B.,Cools Jan,Schwaller Juerg

Publisher

American Association for Cancer Research (AACR)

Subject

Cancer Research,Oncology

Cited by 93 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Pim Kinase Inhibitors Increase Gilteritinib Cytotoxicity in FLT3-ITD Acute Myeloid Leukemia Through GSK-3β Activation and c-Myc and Mcl-1 Proteasomal Degradation;Cancer Research Communications;2024-02-16

2. Targeting Pim kinases in hematological cancers: molecular and clinical review;Molecular Cancer;2023-01-25

3. Non-canonical H3K79me2-dependent pathways promote the survival of MLL-rearranged leukemia;eLife;2021-07-15

4. PP2A-activating Drugs Enhance FLT3 Inhibitor Efficacy through AKT Inhibition–Dependent GSK-3β–Mediated c-Myc and Pim-1 Proteasomal Degradation;Molecular Cancer Therapeutics;2021-02-10

5. Non-canonical H3K79me2-dependent pathways promote the survival of MLL-rearranged leukemia;2020-12-04

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